Genetic methods14 min14 August 2026

From PCR to whole genomes: what each method can detect

A ‘genetic test’ is not one machine. A point variant, repeat expansion, deletion, fusion and methylation change require different methods and quality controls.

TYPE AND BOUNDARYEducational review · assay selection belongs to a qualified laboratory

ONE-MINUTE SUMMARY

  • A broader assay is not automatically more complete.
  • A negative result applies only to variant classes the method can detect.
  • A clinical conclusion needs a validated specimen-to-interpretation process.
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1. PCR and qPCR: a focused question

PCR amplifies a selected DNA region. It can test a known variant, a small target set and some repeat or infectious sequences; qPCR also measures signal over time and can estimate quantity. Its strength is speed and sensitivity, while its central limitation is that it cannot interrogate targets it was not designed to amplify.

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2. Sanger: a short sequence at high accuracy

Sanger sequencing reads a limited region and is useful for one gene, a familial variant or focused confirmation. It scales poorly to hundreds of genes, may miss low-level mosaicism and does not replace methods for large rearrangements or complex repeats.

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3. SNP chips and chromosomal microarray

A genotyping SNP chip queries preselected common sites and is widely used in DTC products and research. Clinical CMA looks for genomic gains and losses and sometimes regions of homozygosity. Neither equals sequencing: an unknown rare variant between probes can remain invisible.

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4. NGS panels: deep, within selected boundaries

A panel sequences tens or hundreds of genes at once, often at high depth, which is useful in inherited disease and oncology. Yet gene lists, exon and intron boundaries, and CNV/fusion algorithms differ. A panel name does not reveal its coverage; the technical specification does.

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5. Exome and genome: broader, not omniscient

WES mainly analyses protein-coding regions, while WGS covers coding and non-coding sequence. Genome sequencing is often more uniform and improves some structural-variant detection, but both have difficult regions, depend on coverage and bioinformatics, and may miss expansions, methylation or mosaicism without specialised analysis.

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6. RNA, FISH, MLPA and karyotyping answer different questions

RNA sequencing measures expression and can detect fusions or splicing effects. FISH visualises selected rearrangements and defined amplification/copy-number patterns, MLPA measures targeted deletions/duplications, and karyotyping detects large chromosomal changes. These methods are not obsolete; they complement DNA NGS where its physics and design are limited.

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7. Methylation, long repeats and long reads

Imprinting disorders and some tumour classifiers require methylation testing. Repeat-expansion disorders need repeat-primed PCR, Southern blot or specialised analysis. Long-read sequencing can resolve repeats, phase and structural variants, but clinical availability and validation remain indication-specific.

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8. The whole laboratory chain determines the result

Specimen, transport, extraction, depth, minimum allele fraction, controls, reference build, classification and confirmation all matter. Tests should be compared by intended clinical use and variant class, not by the largest advertised gene count.

EDITORIAL PASSPORT

Publisher
OOO NPO Nauchnye Tekhnologii
Format
Editorial evidence review
Sources checked
Sources
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Named external review is disclosed only after documented completion. Factual or scientific errors can be reported through the editorial route.

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REFERENCES

Sources behind the review.

  1. NHGRI DNA sequencing
  2. NHGRI human genomic variation
  3. NHGRI microarray technology
  4. NHGRI FISH
  5. AMP/CAP NGS validation guideline
USE BOUNDARY

This review explains evidence and questions for a clinician. It does not interpret an individual DNA file, diagnose or prescribe treatment.